UPSC MainsAGRICULTURE-PAPER-I201215 Marks200 Words
Q14.

Gene for gene hypothesis

How to Approach

This question requires a clear understanding of the Gene-for-Gene hypothesis, its historical context, limitations, and subsequent modifications. The approach should involve defining the hypothesis, explaining its core principles with examples, discussing its significance in plant pathology, and briefly mentioning the challenges it faces and the evolution of understanding in this area. A structured answer incorporating relevant terminology and examples will be crucial for a good score. Emphasis should be on clarity and conciseness within the word limit.

Model Answer

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Introduction

The Gene-for-Gene hypothesis, proposed by Albert H. Sturtevant and E.C. Hewitt in 1940, revolutionized the understanding of plant-pathogen interactions. Initially, plant diseases were perceived as a general, non-specific phenomenon. However, this hypothesis proposed a specific genetic interaction between a resistance gene (R gene) in the plant and a corresponding avirulence gene (avr gene) in the pathogen. This concept laid the foundation for understanding the genetic basis of plant disease resistance and has been pivotal in developing disease-resistant crop varieties, especially in the context of increasing food demand and climate change challenges.

Understanding the Gene-for-Gene Hypothesis

The Gene-for-Gene hypothesis postulates that a plant exhibits resistance to a pathogen only when it possesses a specific resistance (R) gene, and the pathogen carries a corresponding avirulence (avr) gene. The interaction between these genes triggers a defense response in the plant, preventing or reducing disease development. Conversely, if either gene is missing, resistance is absent.

Core Principles and Mechanism

The hypothesis can be summarized as follows:

  • R gene: Located on the plant's genome, it encodes for a receptor protein.
  • avr gene: Located on the pathogen's genome, it encodes for a protein recognized by the R protein.
  • Recognition: When the R protein recognizes the avr protein, it activates plant defense mechanisms, such as the hypersensitive response (HR) – localized cell death to prevent pathogen spread.
  • Specificity: The interaction is highly specific; an R gene only recognizes a particular avr gene.

Historical Significance & Early Examples

The initial formulation of the hypothesis was based on observations of rust diseases on flax and wheat. For example, the interaction between the Magnaporthe oryzae (rice blast fungus) and rice varieties exhibiting resistance is a classic illustration. The R gene ‘Pi-ta’ in rice recognizes the avr-Pi gene in the blast fungus.

Limitations and Subsequent Modifications

While groundbreaking, the Gene-for-Gene hypothesis has limitations:

  • Oversimplification: It doesn't account for complex interactions involving multiple genes on both sides.
  • Pathogen Evolution: Pathogens can evolve new avr genes, overcoming existing resistance (a process called ‘breaking’ resistance).
  • Broad-Spectrum Resistance: Some plants exhibit broad-spectrum resistance, which isn't easily explained by the simple R-avr gene interaction.

These limitations led to the development of the "Bangalore hypothesis" which suggests that R genes recognize conserved elicitors released by pathogens, rather than specific avr genes. This accounts for the broader resistance observed in some cases.

Modern Perspective – Broad-Spectrum Resistance and Gene Pyramiding

Modern plant breeding strategies utilize the principles of the Gene-for-Gene hypothesis, but with a more nuanced understanding. "Gene pyramiding" involves stacking multiple R genes into a single plant variety to provide durable resistance. Furthermore, research focuses on identifying and utilizing broad-spectrum resistance genes.

Concept Description
Gene-for-Gene Hypothesis A plant exhibits resistance only when a specific R gene matches a corresponding avr gene in the pathogen.
Avirulence (avr) gene Gene in a pathogen that allows it to evade plant defenses in the absence of a corresponding R gene.
Hypersensitive Response (HR) Localized cell death in a plant at the site of pathogen infection, triggered by R gene recognition of avr genes.

Conclusion

The Gene-for-Gene hypothesis, while initially simplistic, provided a crucial framework for understanding plant-pathogen interactions and spurred advancements in plant breeding. Its limitations have led to more sophisticated models and strategies, including gene pyramiding and the exploration of broad-spectrum resistance. Continued research in this area remains vital for ensuring food security in the face of evolving pathogen threats and climate change, ensuring sustainable agricultural practices.

Answer Length

This is a comprehensive model answer for learning purposes and may exceed the word limit. In the exam, always adhere to the prescribed word count.

Additional Resources

Key Definitions

Hypersensitive Response (HR)
A localized programmed cell death in plants that occurs at the site of pathogen infection, preventing further spread of the pathogen. It's a crucial component of plant defense triggered by R gene recognition of avr genes.
Bangalore Hypothesis
This hypothesis proposes that R genes recognize conserved elicitors (molecules) released by pathogens, rather than specific avr genes, explaining broad-spectrum resistance observed in some plants.

Key Statistics

Globally, plant diseases cause an estimated 20-30% losses in crop production annually (FAO, 2019). Understanding resistance mechanisms is crucial to mitigate these losses.

Source: FAO (Food and Agriculture Organization of the United Nations)

The development of rice varieties resistant to blast fungus using the Pi-ta gene has saved farmers billions of dollars in pesticide costs and yield losses (IRRI, various reports).

Source: IRRI (International Rice Research Institute)

Examples

Rice and Blast Fungus

The interaction between rice varieties with the ‘Pi-ta’ R gene and <i>Magnaporthe oryzae</i> demonstrates the Gene-for-Gene principle. The ‘Pi-ta’ gene confers resistance to specific strains of the blast fungus possessing the corresponding avr-Pi gene.

Wheat and Stem Rust

Early studies on stem rust in wheat led to the initial formulation of the Gene-for-Gene hypothesis, highlighting the specific interaction between R genes in wheat and avirulence genes in the rust pathogen.

Frequently Asked Questions

What is the difference between the Gene-for-Gene hypothesis and the Bangalore hypothesis?

The Gene-for-Gene hypothesis proposes a specific interaction between R and avr genes. The Bangalore hypothesis suggests R genes recognize conserved elicitors, explaining broad-spectrum resistance.

Why does the Gene-for-Gene hypothesis have limitations?

It simplifies plant-pathogen interactions, doesn't account for multiple genes, and doesn’t explain broad-spectrum resistance, as pathogens can evolve new avr genes.

Topics Covered

BiologyPlant PathologyGeneticsDisease ResistancePlant ImmunityGenetic Interaction