UPSC MainsMEDICAL-SCIENCE-PAPER-II201210 Marks
Q5.

List the group of drugs used to treat MDR-TB.

How to Approach

This question requires a detailed listing of drugs used in the treatment of Multi-Drug Resistant Tuberculosis (MDR-TB). The answer should categorize the drugs based on their function (core, supplemental) and provide specific examples within each category. Mentioning the duration of treatment and potential side effects will enhance the answer. A structured approach, possibly using bullet points or a table, is recommended for clarity. Focus on drugs currently recommended by WHO guidelines (as of knowledge cutoff).

Model Answer

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Introduction

Multi-Drug Resistant Tuberculosis (MDR-TB) arises when *Mycobacterium tuberculosis* develops resistance to at least isoniazid and rifampicin, the two most potent first-line anti-TB drugs. Treatment of MDR-TB is significantly more complex, prolonged (typically 18-24 months), and expensive than drug-susceptible TB. It necessitates a combination of second-line anti-TB drugs, carefully selected based on drug susceptibility testing (DST). The World Health Organization (WHO) provides updated guidelines for MDR-TB treatment regimens, which are constantly evolving based on new evidence and drug availability. This answer will detail the groups of drugs used in treating MDR-TB, adhering to current WHO recommendations as of my knowledge cutoff.

Drug Groups Used in MDR-TB Treatment

The treatment of MDR-TB typically involves a combination of four or more drugs, categorized into core and supplemental groups. The specific regimen is tailored based on the patient’s drug susceptibility testing results.

1. Core Drugs (Always Included)

  • Fluoroquinolones: These are bactericidal drugs that inhibit DNA gyrase and topoisomerase IV, essential for bacterial DNA replication. Examples include:
    • Levofloxacin
    • Moxifloxacin
    • Ciprofloxacin (less preferred due to lower tolerability)
  • Second-Line Injectables: These are potent bactericidal drugs, but are associated with significant side effects.
    • Amikacin
    • Kanamycin
    • Capreomycin
  • Ethionamide/Prothionamide: These drugs inhibit mycolic acid synthesis, a crucial component of the mycobacterial cell wall. Prothionamide is a derivative of ethionamide.
  • Pyrazinamide: While often used in first-line treatment, pyrazinamide can still be effective against some MDR strains and is often included in regimens.

2. Supplemental Drugs (Added Based on DST & Regimen Composition)

  • Cycloserine: Inhibits D-alanine racemase, an enzyme involved in cell wall synthesis.
  • Para-aminosalicylic acid (PAS): Inhibits folate synthesis.
  • Delamanid: A nitro-dihydro-imidazo-oxazole derivative that inhibits mycolic acid synthesis. It is used for extensively drug-resistant TB (XDR-TB) and treatment-intolerant/non-responsive MDR-TB.
  • Bedaquiline: A diarylquinoline that inhibits ATP synthase, an essential enzyme for mycobacterial energy production. Primarily used for XDR-TB and treatment-intolerant/non-responsive MDR-TB.
  • Linezolid: Inhibits bacterial protein synthesis. Often used in XDR-TB regimens.

3. Treatment Duration and Monitoring

A typical MDR-TB treatment course lasts 18-24 months, divided into an intensive phase (typically 6-8 months) and a continuation phase (12-18 months). Regular monitoring for drug toxicity is crucial, especially for aminoglycosides (amikacin, kanamycin) which can cause ototoxicity (hearing loss) and nephrotoxicity (kidney damage). Liver function tests are also essential due to the hepatotoxicity potential of many anti-TB drugs.

4. Newer Regimens & WHO Recommendations (as of knowledge cutoff)

The WHO has been advocating for shorter, all-oral regimens for MDR-TB, aiming to improve treatment adherence and outcomes. The BPaL regimen (Bedaquiline, Pyrazinamide, Linezolid) is a key example, showing promising results in clinical trials. The use of newer drugs like Delamanid and Bedaquiline is increasingly being incorporated into MDR-TB treatment protocols, particularly for patients with XDR-TB or those who fail initial treatment.

Drug Group Examples Mechanism of Action Common Side Effects
Fluoroquinolones Levofloxacin, Moxifloxacin Inhibit DNA gyrase & topoisomerase IV Peripheral neuropathy, tendonitis, QT prolongation
Second-Line Injectables Amikacin, Kanamycin Inhibit protein synthesis Ototoxicity, nephrotoxicity
Ethionamide/Prothionamide Ethionamide, Prothionamide Inhibit mycolic acid synthesis Hepatotoxicity, gastrointestinal upset
Newer Drugs Bedaquiline, Delamanid Inhibit ATP synthase/Mycolic acid synthesis QT prolongation, hepatotoxicity

Conclusion

Treating MDR-TB requires a multifaceted approach utilizing a combination of second-line drugs, guided by drug susceptibility testing and WHO recommendations. While treatment is prolonged and associated with significant side effects, newer drugs and shorter all-oral regimens offer hope for improved outcomes. Continuous monitoring for drug toxicity and adherence to treatment protocols are crucial for successful management of MDR-TB. Further research and development of novel anti-TB drugs are essential to combat the growing threat of drug-resistant tuberculosis globally.

Answer Length

This is a comprehensive model answer for learning purposes and may exceed the word limit. In the exam, always adhere to the prescribed word count.

Additional Resources

Key Definitions

MDR-TB
Multi-Drug Resistant Tuberculosis (MDR-TB) is defined as tuberculosis caused by *Mycobacterium tuberculosis* strains resistant to at least isoniazid and rifampicin, the two most powerful first-line anti-TB drugs.
Drug Susceptibility Testing (DST)
DST is a laboratory process used to determine whether *Mycobacterium tuberculosis* is resistant to specific anti-TB drugs. It is crucial for guiding treatment decisions in MDR-TB cases.

Key Statistics

Globally, an estimated 450,000 new cases of MDR-TB occurred in 2022.

Source: World Health Organization (WHO) Global Tuberculosis Report 2023

According to the WHO, approximately one-third of the world’s estimated 10.6 million new TB cases in 2022 were drug-resistant.

Source: World Health Organization (WHO) Global Tuberculosis Report 2023

Examples

XDR-TB in Estonia

Estonia experienced a severe outbreak of extensively drug-resistant tuberculosis (XDR-TB) in the early 2000s, linked to inadequate treatment and poor infection control in prisons. This outbreak highlighted the challenges of managing drug-resistant TB and the need for robust public health infrastructure.

Frequently Asked Questions

What is the difference between MDR-TB and XDR-TB?

MDR-TB is resistant to at least isoniazid and rifampicin. XDR-TB (Extensively Drug-Resistant Tuberculosis) is MDR-TB that is also resistant to any fluoroquinolone and at least one of three second-line injectable drugs (amikacin, kanamycin, or capreomycin).

Topics Covered

PulmonologyInfectious DiseasesTuberculosisDrug ResistanceTreatment