UPSC MainsZOOLOGY-PAPER-I201220 Marks
Q6.

Give an illustrated account of interaction of neuroendocrine secretions in promoting metamorphosis in an orthopteran and a lepidopteran insect. Mention the type of metamorphosis that these insects undergo.

How to Approach

This question requires a detailed understanding of insect endocrinology and metamorphosis. The answer should begin by defining metamorphosis and differentiating between the types exhibited by orthopterans and lepidopterans. Then, it should delve into the neuroendocrine control of metamorphosis, specifically focusing on the roles of key hormones like ecdysone and juvenile hormone (JH). Illustrative examples within each insect order are crucial. A comparative approach, highlighting similarities and differences in hormonal regulation, will enhance the answer's quality. Structure the answer by first introducing the types of metamorphosis, then detailing the hormonal control in orthopterans, followed by lepidopterans, and finally a comparison.

Model Answer

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Introduction

Metamorphosis, derived from the Greek words meaning “transformation,” refers to the biological process by which an animal physically develops after birth or hatching, involving a conspicuous and relatively sudden change in the animal's body structure through cell growth and differentiation. Insects exhibit two primary types of metamorphosis: incomplete (hemimetabolous) and complete (holometabolous). Orthopterans, such as grasshoppers and cockroaches, undergo incomplete metamorphosis, characterized by gradual changes through nymphal stages. Lepidopterans, including butterflies and moths, exhibit complete metamorphosis, involving distinct larval, pupal, and adult stages. The entire process is orchestrated by a complex interplay of neuroendocrine secretions, primarily ecdysone and juvenile hormone, which regulate molting and differentiation.

Types of Metamorphosis

Before discussing the neuroendocrine control, it’s essential to understand the two types of metamorphosis:

  • Incomplete Metamorphosis (Hemimetabolous): The insect hatches from an egg into a nymph, which resembles the adult but lacks fully developed wings and reproductive organs. The nymph undergoes several molts, gradually developing into the adult form.
  • Complete Metamorphosis (Holometabolous): The insect hatches from an egg into a larva, which is significantly different from the adult in morphology and ecology. The larva undergoes molts, eventually entering a pupal stage, a quiescent, non-feeding stage where dramatic reorganization occurs. Finally, the adult emerges from the pupa.

Neuroendocrine Control in Orthopterans (Incomplete Metamorphosis)

In orthopterans, metamorphosis is regulated primarily by ecdysone, a steroid hormone, and juvenile hormone (JH). The brain neurosecretory cells release prothoracicotropic hormone (PTTH), which stimulates the prothoracic glands to synthesize and release ecdysone.

Mechanism:

  • Low JH levels & Ecdysone surge: When JH levels are low, a surge in ecdysone triggers molting, leading to the next nymphal stage.
  • High JH levels & Ecdysone surge: High levels of JH, maintained during the nymphal stages, prevent the complete differentiation of adult structures during molting. The insect remains in the nymphal stage.
  • Decreasing JH levels: As the insect approaches adulthood, JH production gradually decreases. A final ecdysone surge with low JH levels triggers the molt to the adult stage, allowing for complete differentiation of adult features like wings and reproductive organs.

Example: In a grasshopper, each molt is initiated by an ecdysone peak. The presence of JH ensures that the molt results in a larger nymph, not an adult. When JH levels fall below a critical threshold, the final molt produces the adult grasshopper.

Neuroendocrine Control in Lepidopterans (Complete Metamorphosis)

Lepidopteran metamorphosis is also controlled by ecdysone and JH, but the regulation is more complex due to the presence of the pupal stage.

Mechanism:

  • Larval Stages: High levels of JH maintain the larval state. Ecdysone surges, in the presence of JH, trigger molts between larval instars (stages between molts).
  • Pupation: A critical decline in JH levels, coupled with an ecdysone surge, initiates pupation. The pupa is a non-feeding, relatively immobile stage where extensive tissue remodeling occurs.
  • Adult Emergence: Another ecdysone surge during the pupal stage triggers the emergence of the adult butterfly or moth.

Example: In the silkworm moth (Bombyx mori), JH maintains the larval stages. When JH levels drop, the larva spins a cocoon and enters the pupal stage. A subsequent ecdysone peak initiates the transformation within the pupa, culminating in the emergence of the adult moth.

Comparative Analysis

Feature Orthopterans (Incomplete) Lepidopterans (Complete)
Metamorphosis Type Hemimetabolous Holometabolous
JH Role Maintains nymphal stage; prevents adult differentiation Maintains larval stage; critical for pupation initiation
Ecdysone Role Triggers molting to next nymphal stage or adult Triggers molts between larval instars, pupation, and adult emergence
Pupal Stage Absent Present – a stage of dramatic reorganization

Conclusion

The interaction of neuroendocrine secretions, particularly ecdysone and juvenile hormone, is fundamental to orchestrating metamorphosis in insects. While both orthopterans and lepidopterans utilize these hormones, the timing and levels of their secretion differ significantly, reflecting the distinct developmental pathways of incomplete and complete metamorphosis. Understanding these hormonal controls is crucial for comprehending insect physiology, evolution, and developing strategies for pest management. Further research into the molecular mechanisms underlying hormone signaling during metamorphosis continues to reveal the intricate complexity of insect development.

Answer Length

This is a comprehensive model answer for learning purposes and may exceed the word limit. In the exam, always adhere to the prescribed word count.

Additional Resources

Key Definitions

Prothoracicotropic Hormone (PTTH)
A neurohormone secreted by the brain of insects that stimulates the prothoracic glands to produce ecdysone, initiating molting and metamorphosis.

Key Statistics

Approximately 80% of all known insect species undergo complete metamorphosis (holometabolous development).

Source: Chapman, R. F. (2013). *The Insects: Structure and Function*. Cambridge University Press.

Insecticides account for approximately 16% of the global pesticide market, with a significant portion targeting larval stages of holometabolous insects.

Source: FAOSTAT, 2022 (knowledge cutoff)

Examples

DDT and JH Mimicry

DDT, a historically used insecticide, interfered with insect nervous system function. However, it was also found to mimic juvenile hormone, causing insects to remain in larval or nymphal stages, unable to reproduce, ultimately leading to population decline. This illustrates the critical role of JH in insect reproduction.

Frequently Asked Questions

What is the role of the brain in insect metamorphosis?

The insect brain produces prothoracicotropic hormone (PTTH), which stimulates the prothoracic glands to synthesize ecdysone. Thus, the brain initiates the hormonal cascade that drives metamorphosis.