UPSC MainsMEDICAL-SCIENCE-PAPER-I201320 Marks
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Q14.

Name the folate antagonists used in treatment of bacterial infections and malignancy. Discuss their mechanism of action also.

How to Approach

This question requires a detailed understanding of folate antagonists, their use in both bacterial infections and malignancy, and crucially, their mechanisms of action. The answer should begin by defining folate antagonists and their importance. Then, it should systematically discuss specific drugs used in each context (bacterial infections and malignancy), detailing their mechanisms at a biochemical level. A comparative approach highlighting differences in usage and mechanisms would be beneficial. Finally, potential side effects should be briefly mentioned.

Model Answer

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Introduction

Folate antagonists are a class of drugs that interfere with folate metabolism, a crucial pathway for nucleotide synthesis and, consequently, DNA and RNA production. This interference disrupts cell growth and division, making these drugs valuable in treating rapidly proliferating cells, such as those found in bacterial infections and cancers. Folate is essential for one-carbon transfer reactions vital for purine and pyrimidine synthesis. The disruption of these pathways leads to impaired DNA synthesis, ultimately inhibiting cell proliferation. Understanding the specific mechanisms of these antagonists is crucial for optimizing their therapeutic use and minimizing adverse effects.

Folate Antagonists in Bacterial Infections

The primary folate antagonist used in treating bacterial infections is Trimethoprim. It selectively inhibits bacterial dihydrofolate reductase (DHFR). DHFR is an enzyme responsible for converting dihydrofolate to tetrahydrofolate, a crucial coenzyme in nucleotide synthesis.

  • Mechanism of Action: Trimethoprim binds to bacterial DHFR with a much higher affinity than to mammalian DHFR. This selective inhibition disrupts the synthesis of tetrahydrofolic acid, essential for bacterial DNA and protein synthesis.
  • Spectrum of Activity: Trimethoprim is effective against a broad range of Gram-positive and Gram-negative bacteria.
  • Clinical Uses: Commonly used in the treatment of urinary tract infections (UTIs), respiratory infections, and Pneumocystis jirovecii pneumonia.
  • Synergism: Often combined with sulfamethoxazole (co-trimoxazole) to achieve synergistic antibacterial effects. Sulfamethoxazole inhibits dihydropteroate synthase, another enzyme in the folate synthesis pathway, creating a sequential blockade.

Folate Antagonists in Malignancy

Methotrexate (MTX) is the most widely used folate antagonist in cancer chemotherapy. It exhibits a broader range of mechanisms and applications compared to trimethoprim.

  • Mechanism of Action: Methotrexate is a structural analog of folic acid and acts as a potent inhibitor of DHFR, similar to trimethoprim, but with less selectivity. It binds to and inhibits human DHFR, disrupting nucleotide synthesis and DNA replication in rapidly dividing cancer cells. However, MTX also has other mechanisms. At higher doses, it inhibits thymidylate synthase (TS) and aminoimidazolecarboxamide ribonucleotide transformylase (AICAR transformylase), further disrupting nucleotide synthesis.
  • Clinical Uses: Used in the treatment of various cancers, including leukemia, lymphoma, breast cancer, and osteosarcoma. It's also used in autoimmune diseases like rheumatoid arthritis and psoriasis due to its immunosuppressive effects.
  • Leucovorin Rescue: A key aspect of methotrexate therapy is "leucovorin rescue." Leucovorin (folinic acid) is a reduced folate that bypasses the DHFR block, providing cells with a source of tetrahydrofolate and mitigating the toxic effects of methotrexate on normal cells.

Comparison of Trimethoprim and Methotrexate

Feature Trimethoprim Methotrexate
Primary Target Bacterial DHFR Human DHFR, TS, AICAR Transformylase
Selectivity Highly selective for bacterial DHFR Less selective; affects both bacterial and mammalian DHFR
Main Clinical Use Bacterial Infections Cancer Chemotherapy & Autoimmune Diseases
Rescue Agent Not typically required Leucovorin (Folinic Acid)

Adverse Effects

Both trimethoprim and methotrexate can cause adverse effects. Trimethoprim can cause gastrointestinal disturbances and skin rashes. Methotrexate can cause myelosuppression (bone marrow suppression), mucositis (inflammation of the mucous membranes), and liver toxicity. Regular monitoring of blood counts and liver function is essential during methotrexate therapy.

Conclusion

Folate antagonists, particularly trimethoprim and methotrexate, represent important therapeutic agents in combating bacterial infections and malignancy. Their mechanisms of action, centered around disrupting folate metabolism, effectively target rapidly dividing cells. While trimethoprim offers selective antibacterial activity, methotrexate exhibits broader applications but requires careful monitoring and rescue strategies due to its potential toxicity. Continued research into novel folate antagonists and strategies to enhance their selectivity and efficacy remains crucial for improving patient outcomes.

Answer Length

This is a comprehensive model answer for learning purposes and may exceed the word limit. In the exam, always adhere to the prescribed word count.

Additional Resources

Key Definitions

Dihydrofolate Reductase (DHFR)
An enzyme that catalyzes the reduction of dihydrofolate to tetrahydrofolate, using NADPH as a cofactor. Tetrahydrofolate is essential for one-carbon transfer reactions in nucleotide synthesis.
Synergism
The interaction of two or more agents that produces an effect greater than the sum of their individual effects. Co-trimoxazole (trimethoprim + sulfamethoxazole) exemplifies synergism in antibacterial therapy.

Key Statistics

In 2022, approximately 1.9 million new cancer cases were diagnosed in the United States, with methotrexate being a key component in the treatment of many of these cases.

Source: American Cancer Society, 2023 (Knowledge Cutoff: Dec 2023)

Antibiotic resistance to trimethoprim-sulfamethoxazole has been increasing globally, with resistance rates exceeding 50% in some regions for common urinary tract pathogens.

Source: World Health Organization, Antimicrobial Resistance Global Report on Surveillance, 2023 (Knowledge Cutoff: Dec 2023)

Examples

Methotrexate in Rheumatoid Arthritis

Low-dose methotrexate is a first-line treatment for rheumatoid arthritis. It suppresses the immune system by inhibiting purine synthesis in rapidly dividing lymphocytes, reducing inflammation and joint damage.

Frequently Asked Questions

What is leucovorin rescue and why is it important?

Leucovorin rescue involves administering folinic acid (a reduced folate) after high-dose methotrexate therapy. It bypasses the DHFR block caused by methotrexate, providing normal cells with the tetrahydrofolate they need for survival and mitigating the drug's toxic effects.

Topics Covered

PharmacologyOncologyFolate AntagonistsChemotherapyInfectionsMalignancy