UPSC MainsBOTANY-PAPER-II201415 Marks
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Q9.

When the glucose-grown cells of 'E. Coli' are transferred to the medium containing lactose, what will be the fate of 'Lac Operon'?

How to Approach

This question requires a detailed understanding of the *lac* operon and its regulation in *E. coli*. The answer should explain the initial state of the operon in glucose, the changes occurring upon lactose introduction, the roles of key players like the repressor protein, cAMP-CAP complex, and the resulting gene expression. A step-by-step explanation of the operon’s fate, from induction to full expression, is crucial. The answer should be structured logically, starting with the initial state and progressing through the regulatory mechanisms.

Model Answer

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Introduction

The *lac* operon in *Escherichia coli* is a classic example of an inducible gene regulatory system. It controls the metabolism of lactose, a disaccharide, by encoding enzymes necessary for its uptake and breakdown. In the presence of glucose, *E. coli* preferentially utilizes glucose as an energy source, effectively repressing the expression of the *lac* operon genes. However, when glucose is scarce and lactose is available, the operon is induced, allowing the bacterium to utilize lactose. Understanding the fate of the *lac* operon when cells are transferred from a glucose-rich to a lactose-rich medium is fundamental to comprehending gene regulation in prokaryotes.

Initial State: Glucose Growth

When *E. coli* cells are grown in a glucose-rich medium, the *lac* operon is largely repressed. This repression is primarily due to two mechanisms:

  • Repressor Protein (LacI): The *lacI* gene, located upstream of the operon, constitutively produces the LacI repressor protein. This repressor binds to the operator region of the *lac* operon, physically blocking RNA polymerase from transcribing the structural genes (*lacZ*, *lacY*, and *lacA*).
  • Catabolite Repression: Even if the repressor is not bound, transcription is still low due to catabolite repression. When glucose levels are high, the concentration of cyclic AMP (cAMP) is low. cAMP is required to bind to the Catabolite Activator Protein (CAP), forming a cAMP-CAP complex. This complex enhances RNA polymerase binding to the promoter region of the *lac* operon. With low cAMP, CAP remains inactive, and transcription is inefficient.

Transfer to Lactose Medium: Induction

When glucose-grown *E. coli* cells are transferred to a medium containing lactose, the following events occur:

1. Lactose Uptake and Allolactose Formation

Lactose enters the cell via a passive transporter. A small amount of lactose is converted into allolactose, an isomer of lactose, by the enzyme β-galactosidase (encoded by *lacZ*). Allolactose acts as the inducer.

2. Repressor Inactivation

Allolactose binds to the LacI repressor protein, causing a conformational change. This change renders the repressor unable to bind to the operator region of the *lac* operon. The repressor dissociates from the operator, removing the primary block to transcription.

3. Catabolite Activation (Gradual)

As glucose is absent, the intracellular concentration of cAMP begins to rise. cAMP binds to CAP, forming the cAMP-CAP complex. This complex then binds to a specific site upstream of the *lac* operon promoter, enhancing RNA polymerase binding.

4. Increased Transcription and Translation

With the repressor removed and CAP activated, RNA polymerase can efficiently bind to the promoter and transcribe the *lacZ*, *lacY*, and *lacA* genes. This leads to the production of β-galactosidase, permease, and transacetylase, respectively. These enzymes facilitate lactose uptake and metabolism.

5. Negative Feedback Regulation

As lactose is metabolized, allolactose levels decrease. This leads to the dissociation of allolactose from the repressor, restoring its ability to bind to the operator. As glucose levels remain low, CAP remains activated, maintaining a high level of transcription until lactose is depleted.

Stages of *Lac* Operon Activation

Stage Repressor CAP Transcription
Glucose Present, Lactose Absent Bound to Operator Inactive (low cAMP) Very Low
Lactose Present, Glucose Absent Not Bound to Operator (Allolactose bound) Active (high cAMP) High
Lactose Present, Glucose Present Not Bound to Operator (Allolactose bound) Inactive (low cAMP) Low

Conclusion

In summary, when glucose-grown *E. coli* cells are transferred to a lactose medium, the *lac* operon undergoes a carefully orchestrated induction process. The removal of repression by allolactose, coupled with the activation of transcription by the cAMP-CAP complex, leads to increased expression of the *lac* operon genes. This allows the cells to efficiently utilize lactose as an alternative energy source. The *lac* operon serves as a powerful model for understanding gene regulation and its responsiveness to environmental cues.

Answer Length

This is a comprehensive model answer for learning purposes and may exceed the word limit. In the exam, always adhere to the prescribed word count.

Additional Resources

Key Definitions

Inducible Operon
An inducible operon is a gene cluster that is typically off (repressed) but can be turned on (induced) in the presence of a specific molecule, called an inducer.
Catabolite Repression
Catabolite repression is the inhibition of the metabolism of alternative carbon sources (like lactose) when a preferred carbon source (like glucose) is present.

Key Statistics

The *lac* operon contains approximately 1000 base pairs of DNA and encodes three structural genes: *lacZ*, *lacY*, and *lacA*.

Source: Molecular Biology of the Gene, 7th Edition (Watson et al., 2014)

The rate of lactose hydrolysis by β-galactosidase can reach up to 10^6 molecules per minute per enzyme molecule.

Source: Biochemistry, 5th Edition (Berg et al., 2002)

Examples

Lactose Intolerance

Humans lacking sufficient β-galactosidase (the product of *lacZ*) are lactose intolerant, unable to efficiently digest lactose, leading to gastrointestinal discomfort. This highlights the biological importance of the enzyme encoded by the *lac* operon.

Frequently Asked Questions

What would happen if the *lacI* gene was mutated to produce a non-functional repressor?

If the *lacI* gene was mutated to produce a non-functional repressor, the *lac* operon would be constitutively expressed, even in the presence of glucose and absence of lactose. This is because the repressor protein would be unable to bind to the operator and block transcription.

Topics Covered

BiologyMolecular BiologyGene RegulationOperonsBacteriaMetabolism