UPSC MainsBOTANY-PAPER-II201515 Marks
Q22.

Describe different sub-mitochondrial complexes associated with electron transport chain of aerobic respiration.

How to Approach

This question requires a detailed understanding of the electron transport chain (ETC) and its components. The answer should focus on describing each sub-mitochondrial complex (Complex I-IV, ATP synthase) – their structure, function, electron carriers involved, and proton pumping activity. A clear, organized structure with headings and subheadings is crucial. Mentioning the chemiosmotic theory and its relation to ATP synthesis will add value. Diagrams, while not possible in text format, should be mentally visualized while explaining the process.

Model Answer

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Introduction

Aerobic respiration, the primary pathway for ATP generation in eukaryotes, culminates in the electron transport chain (ETC) located within the inner mitochondrial membrane. This intricate system harnesses the energy from electron carriers (NADH and FADH2) to establish a proton gradient, which subsequently drives ATP synthesis. The ETC isn’t a single entity but a series of four protein complexes (I-IV) and a fifth complex, ATP synthase, working in concert. Understanding the structure and function of each complex is fundamental to comprehending cellular energy production. This answer will detail each of these sub-mitochondrial complexes and their roles in the ETC.

Complex I: NADH-CoQ Oxidoreductase

Also known as NADH dehydrogenase, Complex I is the largest complex in the ETC. It accepts electrons from NADH, oxidizing it to NAD+, and transfers them to ubiquinone (CoQ). This process involves the transfer of two electrons from NADH to FMN (flavin mononucleotide), a prosthetic group within the complex, and then to iron-sulfur (Fe-S) clusters. Crucially, Complex I pumps four protons (H+) from the mitochondrial matrix to the intermembrane space per pair of electrons transferred, contributing to the proton gradient.

Complex II: Succinate-CoQ Oxidoreductase

Complex II, also called succinate dehydrogenase, is unique as it’s also part of the Krebs cycle. It accepts electrons from succinate, oxidizing it to fumarate, and transfers them to ubiquinone (CoQ). Unlike Complex I, Complex II does *not* directly pump protons across the membrane. It contains FAD (flavin adenine dinucleotide) as a prosthetic group, which accepts electrons from succinate. The electrons are then passed through a series of Fe-S clusters to CoQ.

Complex III: CoQ-Cytochrome c Oxidoreductase

Complex III, also known as cytochrome bc1 complex, accepts electrons from ubiquinol (QH2), the reduced form of CoQ, and transfers them to cytochrome c. This process is coupled to proton pumping. Complex III utilizes the Q cycle, a complex mechanism involving the oxidation and reduction of CoQ, to pump protons across the membrane. For every two electrons transferred from QH2 to cytochrome c, Complex III pumps approximately four protons into the intermembrane space.

Complex IV: Cytochrome c Oxidase

Complex IV, or cytochrome c oxidase, is the final protein complex in the ETC. It accepts electrons from cytochrome c and transfers them to molecular oxygen (O2), reducing it to water (H2O). This is the terminal step in the ETC and is essential for sustaining aerobic respiration. Complex IV also pumps protons across the membrane, contributing to the proton gradient. It contains heme groups and copper ions, which are crucial for its catalytic activity. Cytochrome c oxidase is highly regulated and can be inhibited by substances like cyanide, carbon monoxide, and azide.

Complex V: ATP Synthase

ATP synthase, also known as Complex V, is not directly involved in electron transfer but utilizes the proton gradient established by Complexes I, III, and IV to synthesize ATP. It consists of two main components: F0 and F1. F0 is an integral membrane protein that forms a channel allowing protons to flow down their electrochemical gradient from the intermembrane space back into the matrix. This flow of protons drives the rotation of a subunit within F0, which in turn drives the rotation of the F1 subunit. F1 is located in the matrix and catalyzes the phosphorylation of ADP to ATP.

Chemiosmotic Theory

The entire process of electron transport and proton pumping is explained by the chemiosmotic theory proposed by Peter Mitchell in 1961. This theory states that the energy released during electron transport is used to create a proton gradient across the inner mitochondrial membrane. This gradient represents a form of potential energy, known as the proton-motive force, which is then harnessed by ATP synthase to drive ATP synthesis.

Complex Electron Donor Electron Acceptor Proton Pumping
I NADH CoQ 4 H+/2e-
II Succinate CoQ 0 H+/2e-
III CoQH2 Cytochrome c 4 H+/2e-
IV Cytochrome c O2 2 H+/2e-
V H+ gradient ATP N/A (uses gradient)

Conclusion

In conclusion, the electron transport chain is a remarkably efficient system for generating ATP. Each sub-mitochondrial complex plays a crucial, interconnected role in this process, from accepting electrons to pumping protons and ultimately driving ATP synthesis. The chemiosmotic theory provides a unifying framework for understanding how these complexes work together to convert the energy stored in NADH and FADH2 into the readily usable energy of ATP. Disruptions in any of these complexes can have severe consequences for cellular energy production and overall organismal health.

Answer Length

This is a comprehensive model answer for learning purposes and may exceed the word limit. In the exam, always adhere to the prescribed word count.

Additional Resources

Key Statistics

Approximately 30-32 ATP molecules are produced per glucose molecule through oxidative phosphorylation (as of 2023 knowledge cutoff).

Source: Textbooks of Biochemistry and Cell Biology

Approximately 90% of the energy derived from glucose oxidation is captured in the form of ATP through oxidative phosphorylation (as of 2023 knowledge cutoff).

Source: Lehninger Principles of Biochemistry

Examples

Mitochondrial Diseases

Defects in ETC complexes can lead to mitochondrial diseases, characterized by impaired energy production and affecting tissues with high energy demands like muscle and brain. Leigh syndrome is an example of a severe mitochondrial disorder often caused by mutations affecting Complex I.

Frequently Asked Questions

What happens if oxygen is not available in the ETC?

If oxygen is not available, the ETC will stall. Electrons cannot be transferred to the final electron acceptor, leading to a buildup of electrons in the preceding complexes. This ultimately halts ATP production and can lead to the accumulation of reactive oxygen species (ROS).

Topics Covered

BiologyBiochemistryMitochondriaElectron Transport ChainAerobic Respiration